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This role applies specifically to membrane-incorporated PUFA-PLs, as the oxidation of PUFAs that are not membrane anchored and incorporated into PUFA-PLs do not contribute to ferroptosis ( There are two main mechanisms of lipid peroxidation in ferroptosis: enzyme-mediated and auto-oxidation ( vice versa , either promoting or directly contributing to lipid peroxidation ( + ( 2 O 2 ) react resulting in hydroxyl radicals and ferric iron, amongst other products ( When ferroptosis was originally described, it was demonstrated that iron chelation and supplementation decreased and increased ferroptosis, respectively ( 5 Iron and IRPs in ferroptosis Ferroptosis is driven by extensive iron-dependent accumulation of lipid reactive oxygen species (ROS), which ultimately commits cells to death ( It is currently understood that uptake of transferrin-bound iron, via the IRP target TFRC, is necessary for ferroptosis and that RNAi knockdown of TFRC decreases ferroptosis sensitivity (Wang et al

Nanoscale Dynamic Readout of a Chemical Redox Process Using Radicals Coupled with Nitrogen-Vacancy Centers in Nanodiamonds