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Well, heat shock proteins, one of the main things they do is they basically make sure the proteins inside of your cells maintain their proper three-dimensional structure, and are folded right, and so they're not prone to aggregating and forming these plaques in your arteries, and also in the brain

Dependent on the form of ADT used, build-up of steroid precursors occurs that are shunted to the synthesis of other steroids, such as progesterone (e.g., treatment with CYP17A1 inhibitor abiraterone), some of which can stimulate CaP progression via co-occurring accommodating changes in AR [35]

doi: 10.3390/cells11233778