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Under the same exposure protocol, we observed the activation of the NGF/TrkA pathways in the cerebellum ( FIGURE 2 FIGURE 3 These results indicate that systemic treatment with BSO induces the synthesis of NGF by peripheral tissues, as well as the NGF and TrkA protein levels and TrkA phosphorylation in the striatum, possibly as a response to systemic oxidative stress induced by GSH depletion after BSO treatment (Table 2), suggesting that NGF is a redox sensor in both the peripheral tissues and striatum

10.1038/s41586-023-06878-9 Nature 62 FuP.ChenY.WuM.BaoB.YinX.ChenZ.et al (2023)

Furthermore, treatment with LBP suppressed the expression level of calpain-1 and suppressed the NF-B nuclear translocation in myocardium of DCM rats

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