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Fragile X mental retardation protein (FMRP), encoded by the FMR1 gene, negatively regulates the synthesis of post-synaptic glutamate receptors and contributes to synaptic plasticity [20, 21]

For the second generation we repeated the process described for the first generation, and from the 50 best sequences for each group, we selected five diverse sequences as parents for the third generation

The risk of publication bias was assessed quantitatively with the Eggers test [28]

In a DSS-induced colitis mouse model, CS reduced disease activity, ameliorated histopathological alterations, upregulated tight junction proteins, decreased TNF expression, and modulated gut microbial composition by increasing Lactobacillus and Blautia populations (185)