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doi: 10.1016/j.neuron.2008.04.010, PMID: [DOI] [PMC free article] [PubMed] [Google Scholar] Jo S., Kim S

Others have studied active disease directly

Universities such as the University of Rochester and Emory University are contributing to the fundamental understanding of these interactions, potentially paving the way for future therapeutic applications

GABA-T is highly expressed in human glial cells and is responsible for clearing released GABA from the synapses to convert GABA into glutamate, which is then fed into the glutamine pool (Fig