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The nonenzymatic functions of GSTs involve their interactions with cellular proteins, such as, Jun N-terminal kinase,(JNK), tumor necrosis factor receptor-associated factor-2 (TRAF2), apoptosis-signal-regulating kinase 1 (ASK), serine/threonine kinases (PKA, PKC), and tissue transglutaminase 2 (TGM2), during which, either the interacting protein partner undergoes functional alteration or the GST protein itself is post-translationally modified and/or functionally altered [53], [74]

Evidencia (pralsetinib): Un estudio de fase I/II, multicntrico y sin anonimato (ARROW [NCT03037385]) evalu la eficacia e inocuidad del pralsetinib.[38] En el momento lmite para los datos, 147 pacientes tenan CTM con alteracin de RET y 22 pacientes tenan cncer de tiroides diferenciado positivo para una fusin de RET

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