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However, this theory inadequately explains the intricate metabolic and pathological processes of MAFLD, leading to the emergence of the "multiple-hit theory." This model attributes MAFLD progression to numerous parallel factors, including dysregulated adipokines, cytokines, mitochondrial dysfunction, endoplasmic reticulum stress, gut microbiota alterations, lipid metabolism, lipotoxicity, oxidative stress, genetic susceptibility, and epigenetic changes [5]

The reclassification of BPC-157, TB-500, KPV, MOTs-c, and GHK-Cu underscores the FDA's commitment to safety and evidence-based practice