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Mechanism Involving Nrf2-Keap1 Complex Mitigates the NLRP3 Inflammasome in PD In PD, the NLRP3 inflammasome acts as a central driver of neuroinflammation, perpetuating a self-sustaining inflammatory loop that accelerates the degeneration of dopaminergic neurons

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(2020) demonstrated that GHK-Cu protected against bleomycin-induced pulmonary fibrosis in mice via anti-inflammatory mechanisms, reducing fibrotic tissue accumulation and preserving lung function