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Recent findings have indicated that levels of the atypical methyltransferase METTL1 are elevated in cancer cells, thereby promoting glutamine synthase (GLUL) expression in an m6A-dependent manner, which in turn facilitates cancer cell proliferation [44]

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Tuttavia, importante notare che queste osservazioni provengono esclusivamente da studi animali e che i dati di sicurezza nell'uomo rimangono molto limitati

The metabolites produced by the altered intestinal microorganisms, such as SCFA and ethanol, may affect the bile acid and fat metabolism process in the liver, leading to the accumulation of fat, which, in turn, induces hepatic steatosis and triggers the development of Mets ( Figure 2A )