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Weyand, MD, PhD, FAHA Professor of Medicine and Immunology, Mayo Clinic Professor Emerita, Stanford University Cornelia M

Surgical ischemia, trauma, and circulatory perturbations can derail mitochondrial function in immune cells, leading to excessive DAMPs release, inflammasome activation, metabolic failure, and ultimately tissue damage and immunosuppression

Preclinical animal models have demonstrated that inositol supplementation can actively counteract sympathetic nervous system overactivation