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Specifically, gut microbial metabolism of phosphatidylcholine, the major dietary source of choline, was first shown to produce a proatherogenic metabolite, trimethylamine-N-oxide (TMAO), via initial formation of trimethylamine (TMA), followed by host hepatic flavin monooxygenase mediated conversion to TMAO (Wang et al., 2011) (Figure S1

Its derived from the naturally occurring peptide in the bodys gastric juice

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