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By binding to receptors in both the pituitary and hypothalamus, it triggers robust, pulsatile GH release that mimics natural secretion patterns [2]

Several studies have reported that certain mtDNA mutational signatures may be specific to tumor types [73, 124, 481]

These microbial metabolites contribute to damage associated with senescence-associated SASP by increasing the expression of inflammatory molecules such as TNF-, IFN-, IL-1, IL-6, and MMPs, which in turn results in a persistent proinflammatory condition or inflammation (54)

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