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This polarity-dependent decrease in inhibition mediated by ALLO may have important implications for how we approach the memory deficits associated with anxiety disorders, which may prove to be amenable to therapeutic strategies targeting the expression and/or activity of GABA A Rs of the 42 subtype (346)

Celecoxib-Induced Gastrointestinal, Liver and Brain Lesions in Rats, Counteraction by BPC 157 or L-Arginine, Aggravation by L-NAME

These studies should focus on medications likely to be used concurrently in patient populations who might benefit from peptide therapy