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10.3390/ijms19102869 33 Anonymous (1998)

In the cell culture model of Wilsons disease, excessive copper accumulation unleashed a cascade of cellular stress responses, including autophagy, oxidative stress, and inflammation driven in part by the dysregulation of key signalling hubs such as NFKB and AP1

The role of ibrutinib in the actual and future therapeutic algorithm

Nei modelli l'AHK-Cu aumentava l'espressione di VEGF, incrementava la proliferazione dei fibroblasti dermici e la densita capillare perifollicolare, suggerendo un migliore apporto vascolare ai follicoli