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L-Arginine has been shown to restore mitochondrial function in bronchial epithelial cells, improving ATP levels and mitochondrial activity in asthma models (Xu et al., 2016)

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Recently reported, deacetylation based on NAD+ signaling was able to enhance the expression of key proteins implicated in PI3KIII-dependent autophagy or promoted Parkin-independent mitophagy through the liver kinase B1 (LKB1)/AMPK/tuberous sclerosis complex 2 (TSC2) or the SIRT3/FUNDC1 pathways [9]