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Inactive NERNST versions were generated for control purposes by replacing active-site cysteines by serines in both the NTR and Trx domains using site-directed mutagenesis (Supplementary Fig

ARA-290 peptide is sourced by research teams investigating the innate repair receptor a heteromeric receptor complex distinct from the classical erythropoietin receptor and its associated intracellular signaling cascades in controlled in vitro environments

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