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This provided direct evidence of increased lipid peroxidation in the CF airway and strongly suggested that this elevation is a consequence of the pulmonary inflammation characteristic of CF, rather than a direct, inflammation-independent outcome of the primary CF defect

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Additionally, there is a shift towards a low-proliferation phase, and a breakdown of cell junctions and apical-basal polarity, which facilitates tumor cell movement and invasion (Pastushenko and Blanpain, 2019)