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In this context, pro-inflammatory cytokines (e.g., TNF, IL-1, IL-8) can activate NET formation ( In the context of CVD, NETs not only exert cytotoxic and prothrombotic effects (Warnatsch et al., 2015), but also contribute to plaque formation (Silvestre-Roig et al., 2019)

Multidimensional investigation of potential anti-cancer targets of glutamine metabolism and their inhibitors Targeting glutamine metabolism-related enzymes and their inhibitors As indicated by the aforementioned evidence, it can be hypothesised that glutamine-addicted cancer cells are reliant on GLS for survival

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A characteristic dysbiotic signature in inflammatory bowel disease (IBD) involves the expansion of Proteobacteria alongside a reduction in the abundance of bacterial members from the Bacteroidetes and Firmicutes phyla [93]