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Excessive heterochromatinization of mitochondrial gene repeat elements in failing hearts may lead to gene silencing and impair cardiac function

It induces tumor cell necrosis, offering a targeted mechanism for exploring potential therapeutic strategies without harming normal cells

Acknowledgements We are grateful to Tibor Pli and Andrs Dr from the Institute of Biophysics, HUN-REN Biological Research Centre for providing access to the spectrofluorometer Fluorolog 3 (Horiba Jobin Yvon) and to the Malvern Zetasizer Nano ZS device (Malvern Instruments)

The 4-hour GH AUC was higher following the evening injection compared to the morning injection [15]