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Autophagic flux tracing using tandem mRFP-GFP-LC3 probe showed that increase of autophagic flux caused by GSH depletion is augmented by rapamycin and attenuated by autophagic inhibitors 3-MA and Baf-A1 (Fig

Ferroptosis in cancers Ferroptosis and the epithelial-mesenchymal transition In vertebrates, the two basic cell types are epithelial cells and mesenchymal cells, which can transform into one another through a reversible process known as epithelialmesenchymal transition (EMT)