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The mechanisms by which PM 2.5 exposure is thought to exert this effect is through induction of chronic neuroinflammation and oxidative stress, which would rise upon inhalation of PM 2.5 pollutants that deposit in the airways [23,24,25]

Minghui, G

Her respiratory symptoms subsequently improved, but her cognitive issues did not, and an FDG-PET scan showed frontal lobe hypometabolism, with bilateral frontotemporal hypoperfusion and anterior slowing on the EEG