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On the one hand, other SCFAs produced by Clostridium exert immunomodulatory effects in T1D by reducing intestinal permeability (192)

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(5) Notably, this stimulation seems selective for collagen, as non-collagen protein synthesis apparently remained unaltered, suggesting the action may not have been a general support for protein production

It plays an essential role in regenerating and healing injured tissues by upregulating actin and blocking actin polymerization, while also promoting angiogenesis and cell migration [8, 9, 10, 11]