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S2CID 4216672

doi:10.1055/a-0653-7108

In addition to downregulation of injury and inflammation-related pathways, GSEA also revealed suppression of adipogenesis-related pathways in the liver of GPR146-deficient mice, suggesting that GPR146 may promote hepatic steatosis via PPAR-mediated transcriptional programs (Fig

Homozygous UGT1A1*28 causes Gilbert syndrome , a benign condition affecting 5-10% of the population characterized by intermittent unconjugated hyperbilirubinemia (elevated indirect bilirubin) triggered by fasting, stress, illness, or menstruation