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Although IGF2 gene expression is not under GH control, the circulating levels of IGF-II are largely influenced by GH status since IGF-II (as well as IGF-I and IGFBP-3) is rapidly cleared from the circulation if not bound in the ternary complex

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Inhibition of Nod-like receptor family pyrin domain-containing protein 3 (NLRP3) inflammasome activation and the release of IL-1 and IL-18 regulates endothelial cell proliferation and migration, further improving retinal neovascularization and alleviating leakage of impaired vessels [13]