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10.1371/journal.pone.0167854 PLoS One

Beyond these parameters, we suggest a model whereby ROS generation causes prolonged effects on mitochondrial morphology and ROS steady-state levels due to local ROS release from mitochondria into the cytosol, where mitochondrial hyperfusion and motility can lead to cell-wide changes in redox state

To investigate the effect of GHK on astrocytes following ICH, we evaluated the cell viability and cell proliferation in vitro by CCK-8 (Figures 3A,C), cytotoxicity by Lactate dehydrogenase (LDH) (Figure 3B)

These studies suggest that mTOR plays a more complicated role than originally proposed and may function in a context-dependent manner to drive T cell subset differentiation under various pathological or physiological conditions