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Excessive ROS can be reduced by cellular antioxidant systems, such as the Nrf2-mediated antioxidant system, GSH, and NADPH [18, 19]

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In APCs, the MyD88-dependent pathway regulates cell maturation, characterized by the increased expression of CD80, CD86, MHC class II, and inflammatory cytokines, e.g., TNF, interleukin (IL)-6, and IL-12

This non-specific action can also interfere with the physiological signaling roles of ROS (as discussed in Section 2.2), potentially leading to unintended adverse effects or a lack of targeted efficacy