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In cancer, circadian disruptiontriggered by irregular light exposure or shift workhas been linked to increased tumor risk and progression, partly due to dysregulation in clock-controlled ubiquitin-mediated degradation of oncogenes and tumor suppressors such as p53 (Reszka and Zienolddiny, 2018

The classification of healthy astrocytes based on their molecular signatures, together with their specific locations, has only begun to evolveparticularly through lineage-tracing studies of radial glia domains, whose neuronal and astrocyte progeny express remarkably similar homeobox gene sets, migrate along identical routes and form microcircuits 55,56,57

Additionally, variations in IGF expression levels across different stages of AD in humans suggest diverse pathophysiological effects at different disease stages

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